Methods:(1)Set up fetal rat intrauterine distress model.
方法:(1)建立胎鼠宫内窘迫模型。
参考来源 - NOS对宫内窘迫所致胎鼠脑损伤作用机制的研究Objective To explore the feasibility and regularity of neural stem cells (NSCs) culture and inducement differentiation from fetal rat ependyma, and offer the positive control for searching the NSCs seed cells outside the nerve tissue.
目的探寻胚鼠室管膜分离培养及诱导分化为神经干细胞(NSCs)的可行性及规律性,为进一步寻找非神经组织性NSCs种子源提供阳性参照。
参考来源 - 胚鼠室管膜神经干细胞体外诱导分化实验研究·2,447,543篇论文数据，部分数据来源于NoteExpress
Conclusions Fetal rat brain damage is made by LPS-induced intrauterine infection.
结论LPS致宫内感染可造成胎鼠脑损伤。
Conclusion Retinoic acid has a strong and multi-regional teratogenic effect on the skeleton of fetal rat.
结论维甲酸有较强的胎鼠骨骼致畸作用，这种作用是多部位的。
Objective To investigate the effect of ant powder on the brain, liver and placenta of fetal rat with IUGR.
目的了解蚂蚁粉对宫内发育迟缓（IU GR）胎鼠的大脑，肝脏和胎盘的作用。
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